Abstract
Acute kidney injury (AKI) is a common medical condition associated with significant increases in morbidity, mortality, cost of care and risk of chronic kidney disease and end stage kidney disease. Cell cycle arrest is implicated in the pathogenesis and repair process following AKI. The urinary cell-cycle arrest markers tissue inhibitor of metalloproteinase-2 (TIMP2) and insulin-like growth factor binding protein 7 (IGFBP7) have been utilized to predict the risk of AKI in many studies from specific population with good performance.
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