Abstract
Monoclonal immunoglobulin (mIG) is known to show an antibody activity against complement regulating factors, e.g. H, I, C3-convertase resulting in complement alternative pathway deregulation and specific types of renal injury characterized by the absence of intrarenal mIG deposits – thrombotic microangiopathy (TMA) and/or C3-glomerulopathy (C3GP). The analysis of the prevalence, clinical and morphological parameters and long-term renal prognosis in patients with mIG-related TMA or C3GP (mIG-TMA/C3) became the goal of this study.
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