Abstract
Hypertension is the second largest risk factor for disease worldwide1. Up to 10% of cases are drug-refractory;2 these are typified by increased sympathetic outflow.3 This has led to the investigation of sympatholytic neuromodulatory therapies. One potential approach is stimulation of the dorsal root ganglion (DRGS), an established target for chronic pain. The DRG is known to be a source of inter-ganglionic collaterals between the autonomic and somatic nervous systems, raising the possibility of autonomic neuromodulation at this site.
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