Abstract
PREVIOUS work on the oxygen tension in different parts of the placental circulation has been confined almost entirely to sheep and goats, in which the pO2 gradient between the umbilical and uterine circulations is high1 and large changes in foetal pO2 levels are difficult to produce2–4. Thus foetal hypoxia occurs only after drastic reduction in maternal arterial blood pO2, and attempts to raise the foetal pO2 have met with little success despite the administration of pure oxygen to the mother2,4. Even when the foetus is supplied with oxygen by other means, the pO2 gradient between uterine and umbilical vessels is still present3.
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