Abstract
The application of high-density electroanatomic (EA) and functional mapping strategies is a new frontier in ventricular tachycardia (VT) ablation. There is no definitive method to identify arrhythmogenic substrate, and ablation outcomes remain suboptimal. The re-entry vulnerability index (RVI) integrates unipolar activation and repolarisation timing, and low RVI values correlate with vulnerable sites of re-entrant VT origin.
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