Abstract
Long QT syndrome (LQTS) is a cardiac channelopathy characterized by QT prolongation and the potential for ventricular torsadogenic-mediated syncope, sudden cardiac arrest, and sudden cardiac death. Whether patients with LQTS have a primary sinoatrial node (SAN) phenotype of chronotropic insufficiency has been speculated but not demonstrated because of the confounding effects of beta blocker therapy.
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