Abstract
PURPOSE: Intense neutrophilic lung infiltration characterizes severe Pneumocystis carinii pneumonia, with considerable morbidity and mortality attributed to host responses. The P. carinii cell wall constituent β-1,3-glucan has been shown to induce lower respiratory tract inflammatory responses. Our laboratory has also recently documented elaboration of inflammatory cytokines by alveolar epithelial cells in response to P. carinii β-glucan. This investigation explores the signaling events promoting inflammatory cytokine expression from alveolar epithelial cells in P. carinii pneumonia.
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