Abstract
Statins are well known drugs to inhibit cellular cholesterol biosynthesis and widely prescribed to patients with hypercholesterolemia or related cardiovascular conditions. However, the pleiotropic effects of statins on vascular cells functions other than cholesterol management are not fully understood. Phenotypic shifting and migration of vascular smooth muscle cells (VSMCs) play key roles in the progression of atherosclerosis. Phenotypic switching in VSMCs induces the alterations in cell-extracellular matrix (ECM) adhesion and cell migration.
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