Abstract
Platelet activation mediated by multiple signaling pathways and consecutive coronary thrombosis plays a central role in the pathogenesis of acute coronary syndromes [1,2]. Baseline platelet reactivity in these patients is an independent predictor of both successful myocardial reperfusion and the extent of myocardial necrosis after primary angioplasty [3–5]. Dual antiaggregation treatment is the therapeutical mainstay in these patients, but around 15% still develop recurrent thrombotic events [6].
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