Abstract
Spontaneous contrast of the cardiac chambers has previously been reported in patients with poor left ventricular function, myocardial infarction-related left ventricular aneurysms, mitral valve disease and chronic obstructive lung disease. 1–3 The pathophysiology of this phenomenon remains poorly understood and, until recently, red cell aggregation was thought to be the cause. 1,2 Recent experimental studies in animals suggested that platelet aggregation, rather than red cell aggregation, could be involved in this echocardiographic abnormality. 4
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