Abstract
To examine the acute interference of β-blockers with plasma chemokine stromal cell-derived factor-1 (SDF-1), epinephrine and norepinephrine levels, as well as cardiovascular autonomic parameters.
Highlights
Hypertension (HT) is the most prevalent asymptomatic condition affecting nearly 1 billion people worldwide [1,2]
Pharmacologic agents used to treat hypertension and Coronary Artery Disease (CAD) may act in cell signaling pathways involved in major cellular responses such as cell proliferation and differentiation as well as in cell homing [8]
Cell homing is a mechanism that promotes the detection of damaged cells and tissues by undifferentiated stem cells and cell migration, proliferation and differentiation and
Summary
Hypertension (HT) is the most prevalent asymptomatic condition affecting nearly 1 billion people worldwide [1,2]. It is one of the main risk factors of Ischemic Heart Disease (IHD) [3] and commonly diagnosed in primary care; yet, full treatment effectiveness still needs improvement [4]. Journal of Cardiology & Cardiovascular Therapy replacement of apoptotic or necrotic dead cells [7] This process is activated the chemokine stromal cell-derived factor-1 (SDF-1) that is released and binds to its specific receptors CXCR-4 and CXCR-7 located on the surface of stem cells, lymphocytes and neurons [9]. To examine the acute interference of β-blockers with plasma chemokine stromal cell-derived factor-1 (SDF-1), epinephrine and norepinephrine levels, as well as cardiovascular autonomic parameters
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