Abstract
Aim: The evidence supporting the hypothesis that phytosterols act as ligands for LXRs is contradictory. A clear assessment of this hypothesis requires the use of LXRab-deficient animals. Therefore, the aim of the present study was to examine the effects of dietary phytosterol supplementation on intestinal cholesterol absorption and fecal neutral sterol excretion in wild-type and LXRab-deficient mice, as well as the effects of the combined administration of phytosterols and LXRab agonists.
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