Abstract
Preface: Why are Islets so Important? B. Soriat. Stimulus-Secretion Coupling: Are KATP Channels Enough?: Glucose Recognition: Hexose Recognition by the Pancreatic B-Cell and the Gustatory Cell A. Niki, H. Niki. Glucose-Induced Time-Dependent Potentiation and `Run Down' of Insulin Secretion in Islets of Young Rats C.R. Bliss, G.W.G. Sharp. The Role for KATP Channels: ATP-Sensitive K+ Channel Closure Is not an Obligatory Step for Glucose-Induced Priming of Pancreatic B-Cell N. Taguchi et al. Intracellular Messengers: Intracellular Second Messengers S.J.H. Ashcroft. Modulation of Islet Release: Extracellular Messengers for Pancreatic B-Cells T. Yada. B-Cell Metabolism and [Ca2+]: Reciprocal Links between Metabolism and Ionic Events in Islet Cells W.J. Malaisse. B-Cell Heterogeneity. Building an Homogeneous Response from an Heterogeneous Population: Heterogeneity of beta-Cell Secretion: Possible Involvement of KATP Channels M. Faehling, F.M. Ashcroft. IDDM (Insulin Dependent Diabetes mellitus): What Went Wrong? Mechanisms of B-Cell Destruction and Defence: New Aspects to Functioning and Regeneration of Pancreatic beta-Cells: Cyclic ADP-Ribose and Reg Gene H. Okamoto. Pathophysiology of the NIDDM: Islet Growth, Regeneration and Transportation: 52 Additional Articles. Index.
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