Abstract
Cardiac arrhythmias, particularly ventricular tachycardia, have long been recognized as a cause of tubular necrosis, anuria and renal failure.1 The graphic display of phasic, instantaneous blood flow velocity in the renal arteries of unanesthetized, conscious human subjects has afforded an opportunity to study the effect of transient or chronic arrhythmias on the renal circulation.
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Paper version not known
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
