Abstract
Aim: Endocannabinoids are endogenous arachidonic acid-derived lipids binding to cannabinoid receptors CB1 and CB2. Circulating endocannabinoid levels are increased in patients with coronary artery disease, myocardial infarction and chronic heart failure. There is a positive correlation between increased endocannabinoid levels and coronary endothelial dysfunction in obese patients. We aimed to clarify the causal implication of the main endocannabinoid 2-arachidonoyl glycerol (2-AG) in the onset of atherosclerosis.
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