Abstract

Abnormalities in cardiac structure and function are common in patients hospitalized with severe COVID-19 pneumonia who have evidence of myocardial injury based on elevated high-sensitivity cardiac troponin (HScTn).1 Studies performing transthoracic echocardiography (TTE) in an acute setting have consistently demonstrated a high prevalence of right ventricular (RV) dilation and dysfunction, a finding that is associated with early mortality independent of standard clinical and biomarker risk stratification.

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