Abstract
The embryonic explanation for penoscrotal transposition associated with hypospadias is retraction in development of the pars phallica of the urogenital sinus, which is associated with delayed genital tubercle fusion in the midline. Labioscrotal folds fail to migrate caudally, or migrate only incompletely, and they remain fused anterior or lateral to the genital tubercle. In severe forms of prepenile scrotum, other anomalies, e.g. perineal hypospadias-absence of urinary tract, polycystic kidney and imperforate anus-may coexist.
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