Abstract
A quantum leap in managing difficult hypercholesterolemia may be close at hand. Unfortunately, hypercholesterolemia and high cardiovascular (CV) risk have become universal problems throughout the world. Al-Waili et al. in their case report on a mutation in the proprotein convertase subtilisin kexin type 9 serine protease (PCSK9) gene in Omani Arab patients with autosomal dominant familial hypercholesterolemia (ADFH), offered the first description of such a mutation in an Arab population and added emphasis to the possible importance of a new therapeutic approach that appears to be on the horizon.1
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