Abstract
Increasing orbital fatty tissue volume and fatty cell number in patients with thyroid associated ophthalmopathy (TAO) can induce high orbital pressure, eyeball protrusion and visual acuity damage. Orbital fatty tissue as a new endocrine organ can secrete various fatty cell factors, growth factors and protein molecules. Some factors and protein molecules participate in the occurrence and progress of TAO. Therefore, we should pay high attention to study the important role of orbital fatty tissue in TAO.
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