Abstract
PurposeTo study the pathophysiology of the vitreomacular interface disorders (epiretinal membrane, macular hole, vitreomacular traction syndrome).MethodsRetrospective review of published articles concerning the above entities.ResultsPosterior Vitrous Detachement (PVD) variations have been considered to be the initial event leading to vitreomacular interface abnormalities. Depending on the level of PVD (eg schisis, partial or complete), different pathophysiological cataracts may occur involving a number of cells and growth factors at the vitreomacular interface.ConclusionsThe pathophysiology of the vitreomacular interface includes a sequence of events such as cellular migration, proliferation and modulation of the extracellular matrix. The quality and quantity of the involved cells and growth factors vary between different vitromacular interface disorders.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
