Abstract
The review considers mechanisms for the development of respiratory failure caused by lipid metabolic disturbances. It gives data on the lung surfactant composition and lipid imbalance in severe concomitant injury, sepsis, and gestosis. Information on the prenatal and postnatal synthesis of triglycerides and cholesterol is detailed.
Highlights
В обзоре рассматриваются механизмы развития дыхательной недостаточности, обусловленные наруше нием липидного обмена
Human lung surfactant (LS) con sists of lipids by 80—85%, 10—20% and 80—90% of which composed of neutral lipids [5,6,7]
Anaemia in pregnant women is accompanied by disturbances of the phospholipid spectra, an increase of cholesterol and lysophosphatides levels and cer tain changes in the structure of lipid transport sys tem, which provide the renewal of the components of red blood cells (RBC) membranes
Summary
В обзоре рассматриваются механизмы развития дыхательной недостаточности, обусловленные наруше нием липидного обмена. Изменения концентрации липидов крови происходят в различных диапазонах и за висят от этиологии, тяжести заболевания. Одной из возможных причин разницы в концентрациях ТГ и липидов очень низкой плотности (ХС ЛПОНП) при сеп сисе у выживших и умерших больных может быть разница уровней TNF α, ингибирующего актив ность липопротеинлипазы [14]. The changes in blood lipids level occur in different ranges and depend on the etiology and severity of the disease.
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