Abstract
The sympathetics play an important part in the regulation of fetal and neonatal insulin secretion and glucose metabolism during normoxaemia and asphyxia (Dawes 1969). Their humoral effects are well documented: exogenous or endogenous catecholamines, e.g. those released during hypoxaemia and asphyxia, changed both insulin and glucose concentrations as well as glucose consumption, in part through α-adrenoceptor mediated mechanisms (Feige et al. 1976; Bassett 1977; Jones and Ritchie 1978; Jones et al. 1983; Jensen et al. 1987 b). However, even though there is a rich sympathetic innervation of both pancreatic blood vessels and pancreatic islets (Van Campenhout 1927; Simard 1935; Hickson 1970), little is known about indirect or direct neuronal effects of the sympathetics on fetal insulin secretion and glucose metabolism.
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