Abstract
Eighteen experiments pairing the transfer experiment with the kindling paradigm were conducted. The brain homogenate supernatant from rats kindled to clonic convulsions was injected intraperitoneally into naive recipients. Similar material from nonkindled rats was injected into other naive recipients. One, two, two and one-half, and three brain amounts were used. Recipients receiving supernatant from kindled animals were retarded significantly in the development of clonic convulsions for all brain amounts. No clear retardation effect was obtained if the supernatant was injected intracerebrally or if the recipients had reached the convulsion stage.
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