Abstract
Postmortem and in vivo imaging studies have demonstrated atrophy within the basal forebrain cholinergic system (BFCS) in symptomatic Alzheimer’s disease (AD). However, relatively little is known surrounding the relationship between BFCS atrophy and Aβ deposition during preclinical stages of disease. This study explores the relationship between Aβ burden and sub-structural BFCS volume in pre-symptomatic individuals at varying genetic risk for AD.
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