Abstract
Despite the development of various anticancer drugs, anticancer drug resistance from prolonged drug exposure is a major obstacle to cancer treatment. It has been reported that changes in intracellular calcium signals are observed in tumorigenesis. However, the association between anticancer drug resistance and abnormal calcium signaling mechanisms in Non-small cell lung cancer (NSCLC) is not fully understood. The aim of this study is to investigate the relationship between calcium and anticancer activity by observing and controlling altered calcium signals in gefitinib-resistance NSCLC cells and to suggest a fundamental solution to overcome acquired resistance.
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