Abstract
Working memory (WM) impairment contributes to learning difficulties and may be a risk indicator for later mental illness in people with neurodevelopmental disorders like chromosome 22q11.2 deletion syndrome (22q11.2DS). Neural activation associated with WM processing shifts from parietal to frontal regions throughout age in the neurotypically developing brain. In children with 22q11.2DS, however, activation is restricted to the frontal cortex, and parietal development is atypical. The aim was to determine neurocorrelates of WM in children with 22q11.2DS.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
