Abstract
A high fat diet is associated with subtle changes in the gastrointestinal function and in the levels of intestinal messengers secreted from enteroendocrine cells. Enterochromaffin (EC) cells contain serotonin (5-HT) which is a key regulator of gut function. Our recent electrochemical studies have shown that 5-HT levels are increased in a rat model of obesity but the molecular basis for this increased is unknown. We hypothesised that changes in 5-HT synthesis or transport proteins could account for these observations.
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