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Abstract
Purpose: Currently the standard of care for anterior cruciate ligament (ACL) injury in the United States is reconstruction, and while surgery has been demonstrated to improve patient-reported outcomes and restore ligamentous stability, ACL reconstruction has not been found to be protective against posttraumatic osteoarthritis (PTOA). Previous studies have demonstrated that ACL injury triggers a series of inflammatory events that leads to a sustained increase in inflammatory cytokines and biomarkers of cartilage breakdown in synovial fluid, and this process is re-initiated after ACL reconstruction.
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