Abstract
BACKGROUND CONTEXT: Klippel-Feil syndrome (KFS) is a developmental disorder presenting with congenital fusion of at least 2 cervical segments. Due to the altered biomechanics associated with fused segments, previous studies have suggested that KFS patients are possibly predisposed to segmental degenerative and/or stenotic alterations that could directly affect the neural elements. However, a large majority of KFS patients remain asymptomatic. Overall, studies have failed to address the effects that various factors associated with KFS have upon the neural elements, specifically the space available for the cord (SAC), and associated cervical spine-related symptoms.
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