Abstract
Tumor intrinsic immune escape mechanisms have attracted attention as a mechanism of resistance to immune checkpoint inhibitors. We have previously reported that lung cancers overexpressing b-catenin have a poor prognosis due to low infiltration of antigen-presenting cells and lymphocytes into the tumor. Although mechanisms of resistance to immune checkpoint inhibitors by b-catenin have been reported in other cancers, it was not clear whether similar mechanisms exist in non-small cell lung cancer.
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