Abstract
Pre-eclampsia is a disease in pregnancy which contributes significantly to maternal and fetal mortality and morbidity. Moreover, ART-conceived twin pregnancies (Daniel, 2000) and pregnancies after oocyte donation (Yang, 2001) are at higher risk for pre-eclampsia. The etiologic factors of pre-eclampsia have not been completely clear, although evidence supports involvement of genetic, immune, angiogenic, and other mechanisms. To clarify the possible relation between gene expression and the placental dysfunction in pre-eclampsia, we examined the expression of regulated genes in placenta tissues.
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