Abstract
Oxidized LDL in Inflammation: From Bench to Bedside
Highlights
In this special issue, we report how findings regarding oxLDL in inflammation at the benchside can be translated to the bedside and how these observations may help clinical practice
Multiple experimental data carried out in cellular lines and animal models of atherosclerosis support the proatherogenic role of oxidized LDLs through several mechanisms which include chemotactic and proliferating actions on macrophages, stimulation of smooth muscle cells, and eliciting apoptosis
We report how findings regarding oxLDL in inflammation at the benchside can be translated to the bedside and how these observations may help clinical practice
Summary
We report how findings regarding oxLDL in inflammation at the benchside can be translated to the bedside and how these observations may help clinical practice. Ishak Ozel Tekin,[1] AsJm Orem,[2] and Ronit Shiri-Sverdlov[3] Multiple experimental data carried out in cellular lines and animal models of atherosclerosis support the proatherogenic role of oxidized LDLs through several mechanisms which include chemotactic and proliferating actions on macrophages, stimulation of smooth muscle cells, and eliciting apoptosis.
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