Abstract
Objective: Vitamin B12 deficiency due to malabsorption, nutritional deficiency or inborn errors of metabolism may lead to pernicious anemia, subacute combined degeneration (SCD) of the spinal cord and additional neurological symptoms. Randomized prospective studies have shown that pernicious anemia can be safely treated with oral vitamin B12, since trace amounts of oral vitamin B12 are absorbed even in complete absence of intrinsic factor. However, it is still common practice to treat SCD with intramuscular injections. We examplify oral treatment in a case of a 24-year-old female with SCD caused by vitamin B12 deficiency due to a lack of intrinsic factor in chronic lymphocytic pangastritis with marked glandular atrophy.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
