Abstract
Oral health self-care products: realities and myths
Highlights
Mark Wolff: Dental plaque thickness has little to do with its cariogenicity issue; thickness is more closely related to the number of bacterial layers
Summary of the discussions held at the ABOPREV International Symposium ness, this excess, and this volume of bacterial burden for the onset of more severe periodontal disease
Jaime Cury: I would like to talk about the importance of ABOPREV in these discussions
Summary
Ricardo Teles: It is difficult to know whether hormonal changes are causing behavioral changes, leading to gingivitis, or hormonal changes are directly affecting receptors in the gingiva, increasing sensitivity to the effects of plaque. Plaque thickness surely has a direct relationship with gingivitis, with increased risk of attachment loss and development of primarily moderate and localized periodontitis in the areas where an excessive biofilm accumulation is observed. Let us bring in localized juvenile periodontitis to remind us that there are periodontal disease conditions in which no biofilm thickness is needed for the development of severe loss of attachment This does not mean that refractory periodontitis does not have a microbial etiology; strong evidence of this remark lies in the actual response to appropriate antimicrobial therapy These conditions clearly show that there is no need for plaque thickness in order to develop loss of attachment. It is worth mentioning that plaque control is yet the best way to prevent periodontal disease in most individuals, but there are several conditions in which these observations do not work in such a direct and absolute manner, and understanding why these conditions behave in such a different way is an ongoing challenge for periodontitis
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