Abstract
In the problem of the pathogenesis of cardiac decompensation, we are currently interested not only in issues of hemodynamics. It is known that a cardiovascular patient has various metabolic disorders, carbohydrate and protein metabolism suffers, basal metabolism and acid-base balance are disturbed, and undeoxidized metabolic products appear in the blood in excess. In the light of these data, it is of interest to solve the problem of the state of oxidative processes in the tissues of a cardiovascular patient.
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