Abstract

A 57-year-old man presented with sudden onset of leg pain, right-sided weakness, aphasia, confusion, drooling, and severe lactic acidosis (15 mmol/L). He had normal peripheral pulses and demonstrated no pain, pallor, poikilothermia, paresthesia, or paralysis. Empiric antibiotics, aspirin, full-dose enoxaparin, and intravenous fluid were initiated. Lactic acid level decreased to 2.5 mmol/L. The patient was subsequently extubated and was alert and oriented with no complaints of leg or abdominal pain. Unexpectedly, the patient developed cardiac arrest, rebound severe lactic acidosis (8.13 mmol/L), and signs of acute limb ischemia. Emergent computed tomography of the aorta confirmed infrarenal aortoiliac thrombosis. Transient leg pain and transient severe lactic acidosis can be unusual presentations of severe infrarenal aortoiliac stenosis. When in doubt, vascular studies should be implemented without delay to identify this catastrophic diagnosis.

Highlights

  • Acute abdominal aortic occlusion is rare and is associated with high morbidity and mortality rates

  • We present a patient with multi-organ dysfunction, including acute kidney injury, hepatitis, and encephalopathy; associated symptoms included transient leg pain and lactic acidosis, which resolved within the first 24 hours after empiric antibiotics and supportive measures

  • Emergent revascularization was planned but the patient developed pulseless electrical activity/asystole again and passed away. This patient initially presented with sudden onset of leg pain, right-sided weakness, aphasia, confusion, and drooling

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Summary

Introduction

Acute abdominal aortic occlusion is rare and is associated with high morbidity and mortality rates. Patients with severe infrarenal aortic stenosis can present with acute limb ischemia, manifested by sudden pain, pallor, pulselessness, paresthesia, and paralysis (the 5 Ps).

Results
Conclusion

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