Abstract
Mitogen activated protein kinases (MAPKs) respond to noxious stimuli (heat shock, cell stress, and proinflammatory cytokines) by regulating a variety of pathways. MAPKs function by activation or inhibition of proteins via phosphorylation. Spinal cord stimulation using differential target multiplexed programming provided analgesia through modulation of protein and gene expression in a neuropathic pain model. We investigated its role in modulating MAPKs and associated targets in neuropathic pain.
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