Abstract
The Double-Barreled cannon stent-graft Aortic Repair (DoBAR) strategy is a specific method for fixing extremely dilated aortic landing-zone issue. Its clinical validation and application may be examined using computational schemes. Three assumed conditions: the single stent-graft, the longitudinal direction (LD)-type DoBAR, and the sagittal direction (SD)-type DoBAR models were examined by computational fluid dynamics (CFD) simulations. Two specific points in one cardiac cycle were plotted. The non-dimensional frequency parameters were calculated to evaluate the flow field stabilities. The primary axial flow skewed from inner to outer aortic portion after passing through the aortic arch at peak forward-flow time (PFFT) and formed the swing signature at peak reverse-flow time (PRFT). The secondary flow developed as counter-rotating vortices of this model. In LD-type, the septum of antero- posterior chambers separated the primary flow to form two individual axial flows, the morphology resembled as the single model at PFFT. In SD-type, the septum of outer-inner chambers divided the flow pathway into two layers, and then weakened the reversed flow strength at PRFT. The septum of the DoBAR limited the development of secondary flow and the swing pattern disappeared in both types. The Strouhal and Wormersly numbers showed the flow pulsatility intensity decreased with steadier flow in both DoBAR models. The Dean numbers disclose the SD-type had weaker axial velocity and weaker secondary flow
Highlights
Constant high blood pressure can cause damage in the aortic intimal layer and result in a weakened arterial wall
We studied the secondary flow from the aortic arch to the descending aorta, using computational fluid dynamics (CFD) techniques
This study models the rheological properties of aortic blood as sagittal direction-type Double-Barreled cannon stent-graft Aortic Repair (DoBAR) model, which we placed inside the aorta at 90 ̊ to the sagittal plane (Figure 1C)
Summary
Constant high blood pressure can cause damage in the aortic intimal layer and result in a weakened arterial wall. The weakened arterial wall can enlarge and form a bulge sac. When the aortic diameter is more than 1.5 times the normal aortic diameter, it is referred to as an aortic aneurysm. Aneurysms can form in any artery throughout the body. The descending aorta and the infra-renal aorta are two of the most affected structures. Aortic aneurysms can rupture and cause life-threatening internal bleeding. The incidence of a thoracic aortic aneurysm is approximately 5–10 per 100 000 patient years, at least 3%– 4% of which occurs in patients aged more than 65 years old [1,2]
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