Abstract
SummaryBenign Paroxysmal Positional Vertigo is one of the most common causes of dizziness. Its characteristic clinical profile is dizziness at head movements. The main diagnostic maneuver of posterior canal Benign Paroxysmal Positional Vertigo is the Dix-Hallpike test. If the maneuver is positive (vertigo and/or nystagmus), the physician can perform the Epley maneuver on the injured side.AimThis paper aims at checking the number of maneuvers necessary for patients with posterior canal Benign Paroxysmal Positional Vertigo to have a negative Dix-Hallpike test.Materials and methodswe carried out a retrospective analysis of 71 charts of patients with posterior canal Benign Paroxysmal Positional Vertigo, who were treated with the modified Epley maneuver.Study DesignCross-Sectional Retrospective.ResultsWe found that 76.00% of the patients analyzed had the symptoms completely resolved and negative Dix-Hallpike test with a single maneuver.ConclusionBased on our results it is possible to conclude that the number of modified Epley maneuvers is variable depending on the etiology, being that the Benign Paroxysmal Positional Vertigo secondary to the traumatic brain injury needed a greater number of maneuvers for Dix-Hallpike test to become negative.
Highlights
The Benign Paroxysmal Postural Vertigo (BPPV) is one of the most common causes of dizziness[1,2,3]
In cases of a positive maneuver, we indicate the Epley repositioning maneuver on the damaged side[12] and the present work aims at checking the number of maneuvers necessary to have a negative Dix-Hallpike test, according to BPPV etiology
The present study was carried out by the chart analysis of 71 patients with otorhinolaryngological diagnosis of posterior canal Benign Paroxysmal Postural Vertigo (BPPV) treated with the modified Epley maneuver, carried out without the use of a bone vibrator on the mastoid, which were discharged from the Speech and Hearing Therapy Department of the State of São Paulo Public Hospital
Summary
The Benign Paroxysmal Postural Vertigo (BPPV) is one of the most common causes of dizziness[1,2,3]. Its characteristic clinical aspect is vertigo triggered by head movements, such as, for example, bending over, looking upwards, laying down or standing up from bed and roll in bed[4,5]. Pathophysiological theory of cupulolithiasis was described by Schuknecht. Such theory states that otolithic fragments detach from the utricle macula and stick to the semi-circular canal cupule, which stops working as angular acceleration transducer and starts working as linear acceleration transducer[6]. The canaliculo lithiasis theory explains that the fragments do not remain adhered to the semi-circular canal cupula, but rather they float in the endolymph. The patient’s head movement causes these fragments to move and an inadequate stimulation of the canal cupula, generating vertigo symptoms[7]
Sign-in/Register to view highlights & summary Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
