Abstract
Research has observed evidence for bothhypo-(supposedly due to a broken mirror neuron system) andhyper-(thought to be the result of deficits in adaptive control) imitation in autism spectrum disorder (ASD). This work sought to adjudicate between thesefindings using an automatic imitation (AI) paradigm with the novel manipulation of the need to engage adaptive control of imitation.Results demonstrated that ASD participants do not display a specific deficit in AI capability, are able to engage in proactive control of AI, and thatrelative to a well-matched effector condition, AI is not selectively associated with ASD symptom severity. These data cast doubt upon the notion of impairments in imitation or its control in ASD.
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