Abstract
Sodium benzoate is effective for treatment of hyperammonemia caused by inborn errors of urea synthesis) -4 Although previous reports ~, 2 have suggested that relatively large doses of benzoate can be tolerated without toxicity, Sorrie adverse effects, 3,4 including death, were reported at higher doses. At present, however, there is little information concerning the blood elimination kinetics of benzoate. We studied the pharmacokinetics of benzoate in two patients with hyperammonemia.
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