Abstract
Pulmonary edema is likely the most frequent cause of acute respiratory failure in critically ill patients. Clinical recognition of pulmonary edema in the tachypneic patient with hypoxemia and roentgenographic evidence of bilateral, diffuse infiltrates is not difficult. However, an accurate and expedient definition of the type of pulmonary edema is frequently challenging and requires thoughtful analysis of what may be a rapidly changing, and conflicting, data base. The ability to distinguish noncardiac pulmonary edema from cardiac pulmonary edema is vitally important since management, beyond initial stabilization, is radically dissimilar.
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