Abstract
Objectives: Heart transplantation became the most effective treatment for end-stage heart failure. Donors after brain death are currently the only reliable source for cardiac transplants. However, hemodynamic instability and cardiac dysfunction have been demonstrated in brain-dead (BD) donors and this could therefore also affect the graft response after heart transplantation. N-octanoyl dopamine (NOD), a novel dopamine derivate, has been shown to protect tissue against hypothermic preservation. We tested the hypothesis that treatment of the BD donor with NOD improves both donor cardiac and graft function after heart transplantation.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
