Abstract
Immunoglobulin A nephropathy (IgAN) is the most common biopsy-proven glomerulonephritis in the world.1 The disease is caused by reduced number of O-glycans in the hinge region of IgA1 (Gd-IgA1) and sequential formation of IgG auto-antibodies that link Gd-IgA1 forming circulating immune complexes that deposit in the mesangial area of glomeruli.2 IgAN may be associated with other diseases3 as well as idiopathic polycythemia/erythrocytosis observed in some IgAN patients referenced in Table 1.
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