Abstract
The rabies virus can produce marked anatomical abnormalities in dendrites and axons in mice, according to Alan C. Jackson of the University of Manitoba, Winnipeg, Canada, and his collaborators. Despite searching for years, researchers failed to detect rabies-elicited anatomical abnormalities beyond characteristic inflammation of the brain and spinal cord in humans or animals infected with this virus. Now, however, detecting these several localized abnormalities helps to fill in crucial gaps in understanding rabies pathogenesis, and also could help in developing novel therapies. Details of their study appear in the January Journal of Virology (82:513–521).
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