Abstract
Impaired clearance of apoptotic cells has been supposed to be involved in the etiopathogenesis of systemic lupus erythematosus (SLE). Furthermore, antibodies against retroviral proteins can frequently be detected in sera of SLE patients without overt retroviral infections. Decreased levels of serum DNase I activities as well as deficiencies in components of the classical complement pathway are well established to predispose to SLE.
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