Abstract
• Choroid and choriocapillaris are potential important targets of therapy early in AMD . • Macular vasculature pathology, like choriocapillaris flow deficits, can be measured using OCT-A. • Subretinal drusenoid deposits result from biogenesis pathways involved in lipid homeostasis . • How to target (over- or under-express) the HtrA1 gene associated with AMD is controversial. • Optimal gene therapies for AMD depend on retinal delivery site and specificity to retinal cell.
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