Abstract
SummaryRats receiving sulfonic acid resin respond by increasing their urinary output of acid phosphate and ammonia, but not in amounts sufficient to compensate for their fecal alkali loss. Those receiving moderate amounts of potassium resin show little change in acid-base balance, while those on high potassium resin intakes show a partially compensated alkali retention. The net fecal alkali loss amounts to 2.4 meq per g resin fed, but a large part of this represents increased phosphate absorption, presumably due to the binding of calcium and/or magnesium by the resin. There is suggestive evidence that some of the extra alkali appearing in the feces as a result of resin ingestion is not in direct combination with the resin, and this may have some bearing on the problem of neutrality regulation.
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Schedule a callMore From: Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine (New York, N.Y.)
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