Abstract
The Deese-Roediger-McDermott (DRM) task is a typical experimental paradigm used to induce false memory production. Previous investigations using event-related potentials (ERPs) revealed differences in a late negative component, FN400, in healthy subjects performing the DRM. However, false memory production may be related to the earlier engagement of cortical circuits. To date, there is no study exploring the electrophysiological activation of early and late neural networks DRM-related in healthy subjects.
Highlights
The way our memory is able to encode, store and retrieve past events can be influenced or even impaired by cognitive and biological elements [1]
Altogether, our findings indicate that the performance in distinguishing false and true memories is correlated to an early activation of neuronal network
Such activation patterns suggest a more efficient activation of neuronal circuitry allocated to discrimination ability, selective attention and subsequent familiarity processing as well as more efficient prefrontal and parietal cortices activation
Summary
The way our memory is able to encode, store and retrieve past events can be influenced or even impaired by cognitive and biological elements [1]. This is especially relevant in pathological contexts such as schizophrenia where false memories can be associated with symptoms like delusional ideation [1,2,3,4,5,6]. In the study phase of the DRM task, subjects learn lists of words (studied items; e.g. The Deese-Roediger-McDermott (DRM) task is a typical experimental paradigm used to induce false memory production. There is no study exploring the electrophysiological activation of both early and late neural networks DRM-related in healthy subjects
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