Abstract
Developmental changes in the cerebellum of neonatal Long-Evans rats with lead encephalopathy were studied by Golgi technique and by light and electron microscopy. Lead encephalopathy was produced by administering daily doses of lead acetate (600 mg of lead acetate/kg of body weight) through an esophageal catheter. Histopathologic changes were observed at 3, 5, 8, 10, 13, and 15 days of age.
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